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Research highlights the observed activity of GHK-Cu in protecting lungs from fibrosis following bleomycin therapy, suggesting its potential interest as a chemotherapy adjuvant research subject to investigate increased medication doses without exacerbating side effects
It inhibits NNMT (nicotinamide N-methyltransferase), an enzyme regulating metabolism, fat storage, and NAD+ consumption.[4][27][6] NNMT methylates nicotinamide, a NAD+ breakdown product, diverting it from recycling and reducing available NAD+
Digestive tissue recovery becomes more efficient, concluding with the reference to Gut-support of BPC-157
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